An Overview of Gastritis

 An Overview of Gastritis Article


Gastritis is a sickness characterized by the inflammation or perhaps erosion with the lining from the stomach. It is characterized by digestive, gastrointestinal mucosal damage represented by simply inflammation processes, degenerative metaplasia, allergic operations.

Gastritis comes in two forms and perhaps they are:

* Serious Gastritis

It involves the superficial erosion of the gastric mucosa. With acute gastritis, it is self- limiting. Revitalization of the mucosa occurs within just 24 to 72 hours.

* Long-term Gastritis

With chronic gastric pain there is extented and repeated irritation from the mucosa. This results in intensifying, irreversible atrophy of the digestive, gastrointestinal mucosa and glands. It occurs in three forms; * Superficial gastritis: This causes reddened oedematous mucosa with haemorrhage and little erosion. 5. Atrophic gastritis: This happens in all 3 layers of the stomach and is also characterized by a decreased number of parietal and key cells. * Hypertrophic gastritis: Causes a dull and nodular mucosa with irregular thickening rugae. There are two main category; * TYPE A (Fundal): It comes from parietal cellular changes ultimately causing atrophy and cellular infiltration. It may be induced by psycho-emotional stresses. 2. TYPE W (Antral): That occurs in the antrum which is usually as a result of degenerative changes and colonization of the mucosa by bacterias. Example may be the Helicobacter pylori.

With chronic gastritis the complexities are the same as the acute form. Once acute gastric pain is not treated, this progresses in the chronic form.


Serious Gastritis: This really is mostly brought on by excessive alcohol consumption and smoking cigarettes especially by using an empty tummy. Potassium and iron supplements, chronic ingestion of aggravating food or perhaps allergic foods (example mushroom, hot spices or herbs, shellfish etc), ingestion of corrosive poison like business lead, mercury are usually associated with gastritis. Prolonged utilization of non-steroidal anti-inflammatory drugs (NSAIDs) such as acetylsalicylsaure also triggers acute gastric pain.

Gastritis may also develop after major surgical treatment, traumatic personal injury, burns, or severe infections. Gastritis usually occurs in those who have had weight loss surgical procedure resulting in the reconstruction of the digestive tract.

Chronic Gastric pain: This is caused by infection most especially Helicobacter pylori; endotoxins released via infecting bacterias such as streptococci, staphylococci; certain diseases including Crohn's disease; pernicious anemia; chronic bile reflux; stress; excessive the radiation use; chemotherapy and some autoimmune disorders.

Mucous gland metaplasia could also cause this form of Gastritis.


The stomach's rich blood supply, mucous level, and acid environment offer a formidable hurdle to localized infections, as well as the digestive chemicals it creates normally HCL and pepsin. The pathogenesis of gastric pain however , is definitely multifactorial and results from a great imbalance with the aggressive gastric luminal factors, acid and pepsin, and defensive mucosal barrier features of the nasal mucus and bicarbonate. Infections of any type of bacterias called Helicobacter pylori colonize the profound layers of gastric mucosa and weaken its defense system by minimizing the width of the mucosal layer and diminishing mucosal blood flow. This kind of results in the development of gastritis in infected individuals.

Mucous gland metaplasia (a inversible replacement of differentiated cells) takes place in the setting of severe damage with the gastric glands, which then spend away (atrophic gastritis). Intestinal metaplasia (complete or incomplete) typically starts in response to chronic mucosal injury inside the antrum, and...

References: 1 ) Brunner and Suddarth's (11th edition) Textbook of As well as Surgical Medical

installment payments on your Roger T. Malseed, PhD, Springhouse Nurse's Drug Guideline, 5th male impotence. Lippincot, Phila., 2004; 149, 534, 849-852, 1227-1228

a few. Taylor Gollan SW. Stomach Emergencies, next ed. Lippincot, Philadelphia, 1997; 219

four. Weller W. F (2001), Bailliere's Rns Dictionary, 23RD Edition, Harcourt publishers Limited U. E.

Websites consulted:





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